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Additionally, the excess iron ions metabolized by microglia are expelled through the transferrin receptor system, leading to a significant accumulation of iron in neurons (55)
10.3321/j.issn.0376-2491.2008.32.004 62 LongJ
This paper identifies the ERMES
We, therefore, predict that GSH will be required to prevent FA toxicity in Fanconi Anaemia by limiting endogenous free FA level
