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The treatment of tumors formed by the radio-resistant GSCs with an IGF1R inhibitor results in a marked increase in radio sensitivity, suggesting that the blockade of IGF1R signaling is an effective strategy to reverse radio-resistance (Osuka et al., 2013)

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GHK-Cu moduleert meer dan 4.000 genen waaronder collageen synthese, antioxidant enzymen en DNA-herstel

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Histological analysis on day 7 revealed a significant decrease in the expression of proinflammatory cytokines in the wound, including TNF- and IL-6 in the D-CuP groups, accompanied by an increase in IL-10 expression (Fig