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Further, although antioxidant enzyme activity was not directly measured in this study, the cGMP-mediated upregulation of GPx-1 and catalase consequently enhanced protection against hydrogen peroxide-induced cell death (339)
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ROS can be regulated by glutathione peroxidases (GPxs), which are the key enzymes that maintain ROS homeostasis in vivo through the reduction of ROS [10]
The aberrant NF-B signaling and expression of BARTs form an autoregulatory loop for maintaining EBV latency in NPC cells
