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Furthermore, we know that the longer anemia goes untreated, the more neuropsychiatric problems and severity will arise (in an uncertain time frame, shorter or longer)

Therefore, any disorder in these redox/defense systems accelerates lipid peroxidation and facilitates the onset of ferroptosis (Figure 2)

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In thisstudy, we obtained gmhA mutants carrying deletions of the cysB and cysE genes that regulate cysteine homeostasis, cysteine transport ( eamA ), and overexpress the cystine transport gene ( tcyP ), as well as deletions of gshA and gshB glutathione biosynthesis genes (Fig

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