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If a significant deficiency is left untreated over time, some of this nerve damage can become permanent, which is why prompt evaluation matters

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doi: 10.1039/D2FO00478J

Then using a H 2 O 2 -induced senescent TM3 Leydig cell model, we found that FOXO4 maintains the viability of senescent Leydig cells and that the FOXO4 blocker FOXO4-DRI induces apoptosis in these senescent cells

doi: 10.1002/glia.20557, 27 BrownA
