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3 Clinical studies, remaining questions, and future directions Clinical trials of clemastine began in multiple sclerosis patients in 2014 (Green et al., 2017)
Endogenous purines primarily originate from dead cells, and mitochondrial dysregulation leads to an accumulation of purines
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doi: 10.1016/j.neuron.2022.10.020
Neomycin resulted in a robust increase in JNK phosphorylation, and this increase was inhibited by celastrol
