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Genetic basis The possibility of diagnosing multiple members from same family with CFS implies the contribution of genetic factors to CFS risk (Walsh et al

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By exploring how redox signaling via GSH influences inflammatory immune phenotypes across different disease states, we will isolate possible therapeutic interventions for treatment of these conditions

G6PD deficiency reduces the amount of NADPH, which is required for the formation of nitric oxide (NO) [10, 11]

doi: 10.3389/fpsyt.2020.561998 Received 14 May 2020 Accepted 27 October 2020 Published 20 November 2020 Volume 11 - 2020 Edited by Trevor Ronald Norman, The University of Melbourne, Australia Reviewed by Ahmed Nadeem, King Saud University, Saudi Arabia