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For a complete overview of AOD-9604 including reconstitution, storage, and the full preclinical dataset, see the AOD-9604 complete guide

In mouse models of highfat dietinduced liver steatosis, it significantly inhibits hepatic NNMT activity, reduces NAM methylation, increases NAD+ and SAM levels, enhances mitochondrial fatty acid oxidation, reduces hepatic triglyceride and lipid accumulation, lowers lipotoxicity markers (e.g., malondialdehyde, transaminases), improves hepatocellular injury, and reverses steatosis

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Our body uses it for brain health, red blood cell production, and energy production

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