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Knockout of Krppel-like factor 14 (KLF14) and Krppel-like factor 15 (KLF15) in renal tubular cells, as well as activation of activating transcription factor 6 (ATF6) overexpression, can inhibit PPAR, reduce FAO mitochondrial activity, lead to lipid accumulation and insufficient energy supply in renal tubular cells, increase apoptosis, and ultimately cause lipotoxicity-induced RF [45,46,47]

Novel approaches utilising red-shifted dyes to reduce autofluorescence and to allow multiplexing of samples are also emerging

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PMID: 40015949 Light Sci Appl2022 Nov 11;11(1):324