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In vitro studies performed in cultured endothelial progenitor cells (EPCs) showed that TMAO promoted cellular inflammation and increased oxidative stress ( The NF-B pathway plays a regulatory role in the expression of many AS-related pro-inflammatory genes (Tak and Firestein, 2001

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To address this issue, genome-wide association studies were completed to attempt to resolve if HDL-C is a risk index or causal factor

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