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Under normal physiological conditions, intracellular OS results from the abnormal production of ROS during typical mitochondrial respiration

doi: 10.3389/fphys.2017.00595

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Atpenins, potent and specific inhibitors of mitochondrial complex II (succinate-ubiquinone oxidoreductase)

Antioxidant capacity in the lung is substantially reduced and oxidative stress can persist long after the cessation of cigarette smoking or an acute exacerbation of COPD owing to continued production of ROS from endogenous sources [9, 12]