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Neurotoxicity was almost completely prevented by the presence of either NMDA receptor antagonists, MK-801 and D(-)-2-amino-5-phosphonopentanoic acid (D-AP5), or mGluR antagonist, (S)-4-carboxyphenylglycine (S-4CPG)
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Genistein, through its activation of ER, stimulates these processes, counteracting mitochondrial dysfunction observed in BMMSCs exposed to oxidative stress
TB-500 is a synthetic form of Thymosin Beta 4 and is frequently investigated within experimental models examining cellular signalling mechanisms and peptide-regulated biological processes
