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This includes alteration of glutamate transporters as well as direction of glutamate transport, trafficking of Ca 2+ -permeable AMPARs and NMDARs, elevation of glutaminase activity, and suppression of mitochondrial function with alteration of mitochondrial energy generation, thus enhancing excitotoxicity [Figure 5]

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doi: 10.1016/j.nbt.2015.12.005 128 PrasadV