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This phenomenon is due to the upregulation of the expression of glutamine transporters or enzymes by oncogenes such as c-MYC, which accelerate the uptake and metabolism rate of glutamine and then drive the TCA and biosynthetic processes [3, 16] (Fig

Cazzola M, Page CP, Calzetta L, Matera MG

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doi: 10.1007/s00253-001-0902-7

Females of this mouse genotype were clearly more resistant than male rats to methionine deficiency
