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Pre-menopausal women are protected from gout by the hormone, oestrogen
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GPCRs are brought into an active conformational state by interacting with exogenous or endogenous agonists, which then affect the intracellular binding of G-proteins or arrestin proteins
Additionally, it facilitates the infiltration of macrophages and fibroblasts, further emphasizing its therapeutic potential in managing wounds and accelerating the healing process of damaged tissues
