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Results from animal models and some clinical studies highlight the potential of antiplatelet therapies in the preservation of renal function in the context of AKI, but as current strategies also affect other cell types and non-platelet-derived mediators, additional studies are required to further elucidate the extent of platelet contribution to the pathology of AKI and to determine the best therapeutic approach by which to specifically target related pathogenic pathways

Each albumin fusion presented different receptor affinities, correlating to its unfused variants

H.MousaA

This interaction leads to the formation of the Rev-CRM1-Ras-related nuclear protein (Ran)-guanosine triphosphate (GTP) complex, established through coupling with the GTP-bound form of Ran, a transport cofactor

Wellen, C