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A persistent stimulation of endometriotic tissues with TGF-1 was able to reduce NR4A1 activity through AKT-dependent phosphorylation, promoting fibrogenesis in OMA [34] and the treatment with Csn-B, an NR4A1 agonist, markedly decreased the expression of fibrotic markers in vitro and inhibited fibrogenesis in urine endometriosis models, suggesting a new potential target for the treatment of this disease [34]

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In short, our bodies age as a whole, not in isolated parts, and back pain usually has multiple contributing factors

Apoptosis 26 , 431446 (2021)

Conventional treatment vs peptides Before we look at the potential of joint healing peptides, we need to understand the conventional options