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In this study, scientists tried to identify the resistance to toxins of young cells, compared to those of older cells

This specifically reversed the initiated multicausal noxious circuit of the occlusion/occlusion-like syndrome as a general effect

A genome-wide CRISPR/Cas9 screening found that the loss of Kelch-like ECH-associated protein 1 (KEAP1) (464) and SGOL1 (465) promote drug resistance in cancer cells, and the CRISPR-induced deletion of SLFN11 (466), BAK (467), and any CST complex members (468) can initiate drug resistance in cancer cells

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