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Description
Loss of the tumor suppressor LKB1 promotes metabolic reprogramming of cancer cells via HIF-1
Eur J Ther
4d, h, Supplementary Fig
Cellular GSH status in RPE cells is decreased, which has been confirmed in many studies [50,51,62,63,64,65,66]
28-30 These actions are mainly mediated by 2-adrenergic receptor subtypes 29,31 although other adrenergic-receptor-independent mechanisms, 28 vagomimetic, and humoral pathways contribute to the anti-inflammatory effect
