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Pre-existing mitochondrial dysfunction due to aging, diabetes, or ischemia creates a permissive environment for cuproptosis by depleting GSH and reducing SOD2 activity, thereby compromising the organelle's ability to chelate copper and neutralize mtROS [145, 146]

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Vitamin B12 is responsible for making red blood cells, white blood cells, and platelets

Additive side effects remain theoretically possible, where both peptides contribute to similar adverse effects