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On one hand, excessive free radicals and ROS accumulate in the body, causing damage to cellular structures, including proteins, lipids, and nucleic acids, leading to gene mutations that promote the formation and proliferation of cancer cells [107,108,109]
Preparation and in vitro sustained-release delivery properties of CMH@lip@Res-TCeO 2 CMH@lip@Res-TCeO 2 was fabricated by embedding Lipo@Res-TCeO 2 liposomes into CMH2 hydrogel, as illustrated in Fig
Cell death caused by selenium deficiency and protective effect of antioxidants
doi: 10.1186/s13024-021-00464-1 312 SheehanP
Our results suggest that CBS of the TSS pathway plays an important role in maintaining cysteine levels under restricted cystine availability or excess hemin exposure, and CBS upregulation by DMF increases the cellular glutathione levels to protect against ROS and ferroptosis stress in SCD
