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doi: 10.3109/10428199509051709

Mitochondrial-mediated ROS cause oxidative damage to cellular structures, particularly mitochondrial DNA, somatic mutations, genome instability, inactivation of tumor suppressor genes, alterations in metabolic and signaling pathways with simultaneous activation of compensatory antioxidant mechanisms, all of which contribute to cell transformation and thus promote various types of cancer [42]

10.1146/annurev.py.33.090195.001503 9 BarnaB.dmA.KirlyZ

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GHK-Cu enhances the function and production of the dermal fibroblasts, and is still effective even when the cells have been compromised through ageing or damage