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In addition, double knockouts of PGC-1 and PGC-1 in skeletal muscle or the adult heart did not result in a loss in mitochondrial mass, which indicated that PRC may play a role in mitobiogenesis in the absence of PGC-1 and PGC-1 [71, 72], a finding that warrants further investigation

The selective elimination of senescent cells or support for the body in this process through the modulation of autophagy in them, coupled with limiting the impact of senescent cells on their environment, may prove highly effective in anti-cancer therapy [9]