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Treatment with FIN56 significantly reduced the survival of primary and paracrine senescent cells, while TRX-CBI showed cytotoxicity against senescent cells at low concentrations, with minimal effects on non-senescent cells [135]

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The main feature of NAFLD is the FA accumulation in liver parenchyma, due to an enhancement in FA uptake from plasma, in addition to an overstimulation of the de novo lipogenesis via SREBP-1c transcription factor activation [83]