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Sechi et al

The results suggest that GMGS may help define a unique proliferative population of intratumoral CD8 + T cells distinct from conventional exhausted-like or stem-like T cells, potentially associating with therapeutic response of immunotherapy

Dysregulation of C/EBPalpha by Mutant Huntingtin Causes the Urea Cycle Deficiency in Huntington's Disease. Human Molecular Genetics 16: 483498

These include GSH, GSSG, glutathione S -conjugates, -glutamyl amino acids, and -glutamyl p -nitroanilide (a substrate commonly used to assay enzyme activity) (Orlowski and Meister, 1963, Orlowski and Meister, 1970, Wickham et al., 2011)

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