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The expression of the VD metabolism genes CYP2R1 and CYP27A1 (25-hydroxylase), CYP27B1 (1--hydroxylase), and vitamin D receptor (VDR) were downregulated in the livers of mice fed an HFD (GSH- deficient) compared with control diet-fed group

Further, PS exposure of uninfected erythrocytes and increased levels of anti-PS auto-antibodies have been proposed as key mechanisms leading to the loss of non-infected erythrocytes, therefore contributing to malarial anemia (Totino et al., 2010
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We also thank Ana Garca-Quiralte and Babette Mc Cafee for technical assistance
