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Using an integrative approach comprising quantitative proteomics, metabolic mapping via stable isotope 13 C-labeled energy substrates and gas chromatography coupled to mass spectrometry, as well as live-cell bioenergetics analysis, we have identified a constellation of metabolic changes that lead us to conclusively identify two major enzymes linking neurotransmitter and energy metabolism as crucial players in FTD3 pathology

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Adverse event (AE) data were collected throughout the study

Excess intracellular iron is typically sequestered within the ferritin protein, which consists of two subunits: FTH1 and FTL (Yang and Stockwell, 2008)

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