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& Zhou, S.-F

(2005) used an adenovirus-delivered small interfering RNA (siRNA) method to observe the knockdown of c-Met on tumorigenic growth of HCC in in vitro and in vivo trials In the in vitro trial, compared with adenovirus alcohol dehydrogenase (AdH1)-null or mock-infected cells, proliferation of MHCC97-L cells, which had high c-Met expression, were inhibited by adenovirus AdH1-siRNA, and c-Met expression also decreased

Several studies pointed out that GSH and the enzymes associated with the GSH pathway are involved in SARS-CoV-2 infection and COVID-19 disease

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Adequate antioxidant defenses, including optimal glutathione status, are essential for protecting brain cells from this oxidative burden