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Understanding these mechanisms could inform new strategies to prevent metastasis and improve patient outcomes (120)

GSH forms adducts via a nucleophilic substitution reaction mechanism to cisplatin (to its intracellular form, platinum (II) aqua form) via a molar ratio of 2:1, as shown by mass spectrometry, both in vitro and inside leukemic cells [115]

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Cells with a high expression profile of ACSL4 and LPCAT3 have a higher level of PUFA-containing phospholipids, predisposing them to ferroptotic damage due to the reactivity of these lipids toward oxidative stress (61)