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For instance, lipopolysaccharide (LPS)-stimulated microglia can induce astrocytes to release soluble toxic factors that directly promote neuronal and oligodendrocyte death

6 Collectively, these functional changes are consistent with neutrophil polarization toward the N1 proinflammatory phenotype, 7 underscoring the propensity of neutrophils to undergo such polarization within the BM microenvironment remodeled by chronic inflammation

The exact mechanism by which vitamin D improves lipid markers is still under debate 6

In terms of stress responses, AtGSTL1 is strongly up-regulated in several conditions including biotic interactions, treatment with chemicals, salt and iron-starvation stresses and, to a lesser extent, in response to hypoxia

A.JlligM.et al