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The twice-daily administration mode (bid): Mice were given MCTR1 (200 ng/mice, iv) 0.5 h before being subjected to CLP, and then an additional MCTR1 (200 ng/mice, iv) was given 12 h after CLP

Further analyses showed that FKA induced apoptosis mostly via intrinsic pathway as documented by activation of caspase-9 and -3, increase in Bax/Bcl-xL ratio, decrease in mitochondrial membrane potential and release of cytochrome c into cytosol

As, in a mouse model of gastric cancer, lipid aggregation in tumor-associated macrophages (TAMs) upregulated the expression of phosphoinositide 3-kinase (PI3K-) and promoted TAM polarization to the M2 phenotype

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I even ate chips because I love the crunchy sound they make