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c- Jun -N Terminal Kinase-Mediated Degradation of -Glutamylcysteine Ligase Catalytic Subunit Inhibits GSH Recovery After Acetaminophen Treatment: Role in Sustaining JNK Activation and Liver Injury

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The Fok I polymorphism is functionally relevant because it alters the translation initiation site of the receptor, producing either a shorter (424 amino acids) or longer (427 amino acids) VDR protein
