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In addition, peripheral nerve injury enhances spinal neuronal excitability through glutamate receptor activation, coupled with downregulation of spinal glutamate transporter proteins and reduced activation thresholds, leading to increased neuronal excitability and neurotoxicity [38]

In humans, delayed maturation of the antibody response, characterized by a paucity of FcR-activating antibodies, correlates with more severe disease 57

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