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When released (e.g., with vesicle depletion by reserpine or amphetamine) [69,70,152] it can substitute disturbed dopamine (i.e., shortage (reserpine) [69] or excess (amphetamine) [70]) as competing successfully for sites at dopamine receptors (i.e., counteracting the adverse effects of both dopamine agonists [70,152,153] and dopamine antagonists [68,70,152])

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Protects Against Mitochondrial Dysfunction: SS-31 preserves mitochondrial function even under stress, making it a potential therapeutic tool in both medicine and performance applications

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A typical approach is 4 to 12 weeks on, then an off-cycle of equivalent length

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