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5G), suggesting that interference with glutathione metabolism impairs Venetoclax efflux

Understanding the contribution of metabolism to Mycobacterium tuberculosis drug tolerance

This selectivity reduces the risk of the side effects associated with non-selective serotonin elevation

Receptor-mediated signaling cascades also amplify proinflammatory responses through NFB-mediating activation of inflammasome, facilitating the secretion of IL-1 and IL-18 [1]

Importantly, microbial adaptation within oxidative and hypoxic wound microenvironments is increasingly recognized as a driver of antibiotic resistance and delayed tissue regeneration, highlighting the therapeutic advantage of redox-responsive scaffolds capable of modulating local ROS dynamics and infection progression