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In OA, MSCs move to injury sites from subchondral bone, becoming chondrocytes and osteoblasts for tissue repair

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Similarly, activation of Nrf2 was also demonstrated in an in vivo cerebral ischemia model, with increased SOD1 in the nucleus, SOD2, glutathione S-transferase and peroxidase in the cortex, and increased activity of catalase in the striatum, evidencing the protective effects of DATS [157]

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