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The most severe symptoms associated with biotin deficiency and profound biotinidase gene defects are the result of the accumulation of toxic metabolic intermediates

While inhibiting TRAb variations may contribute to hypothyroid phenotypes, stimulating TRAb binds to and activates the TSH receptor, resulting in hyperthyroidism and extrathyroidal characteristics such orbitopathy

Data availability The authors declare that all data supporting the findings of this study are provided in the Manuscript and its Supplementary Information/Source Data files

Comput Biol Chem

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