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In recent years, it has become evident that (a) fungal ROS systems might not play the predicted crucial role in pathogenicity, (b) fungal pathogens, especially necrotrophs, can actively contribute to ROS levels in plants and even exploit the hosts response, and (c) fungi possess superoxide-generating NADPH oxidases similar to mammalian NOX complexes, which are important for pathogenicity [42, 43]

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These patients exhibit lower numbers of retrieved oocytes, lower quality of oocytes, lower number of embryos and quality of embryos, and lower rates of blastocyst formation (118120)

doi: 10.1099/0022-1317-75-10-2595