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In HK-2 kidney cells, PQQ (100 nM) modulates inflammation and senescence by reducing reactive oxygen species (ROS) production and influencing the Keap1/Nrf2 pathway, a crucial antioxidant defense mechanism in cells [66]
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These findings suggest t that apoptosis is closely linked to senescence, with mitochondria playing a key role in coordinating senescence-associated apoptosis
