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SPIGFD in these patients were defined by phenotypic and biochemical characteristics and they were treated with recombinant human (rh)IGF-1 in one of the clinical trials leading to approval of this therapy, as described later

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8, an ultra-shortlived oxidant that inflicts site-specific macromolecular damageDNA base modifications and strand breaks near metal centers, protein carbonylation and initiation of lipid peroxidationwith risk amplified by iron mobilization (e.g., ferritinophagy) and disruption of FeS clusters (Zhang et al