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Description
show that mTOR can regulate signaling through the insulin-like growth factor 1 receptor and that it possesses a new enzymatic activity the ability to phosphorylate proteins on tyrosine residues.
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Awosemo, O., et al
In particular, GSDME-deficiency significantly attenuated the hippocampal atrophy and changed levels of DAM markers in GCLC-KO mice
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