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3A) and nitrotyrosine (Fig
In contrast, mutations in VIT-1 and VIT-2, which are two major apolipoproteins for cholesterol trafficking between tissues, and in HEH-1, an ortholog of human NPC2, which is a soluble luminal protein in lysosomes and responsible for transferring cholesterol to human NPC1 (39, 58), hindered the effects of high cholesterol as ncr-1(0) (Fig 4D)
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doi: 10.1016/0006-291X(89)91360-0
