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Excessive ROS levels can induce cellular damage, initiating a cascade of effects that potentially culminate in tissue and organ injury ( 3.1.1 Impact of ROS on cellular function and atherosclerosis Elevated ROS can induce endothelial cell dysfunction or death, disrupting the vascular barrier and leading to vasoconstrictive and vasodilatory dysfunction, vascular smooth muscle cell proliferation and migration, inflammatory responses, and thrombosisprocesses intimately associated with various cardiovascular disorders, including atherosclerosis, hypertension, and CHD ( in vivo ROS generation represents a promising therapeutic strategy for CHD treatment

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Metformin induces apoptosis via the downregulation of the downstream targets of K-RAS in human A549 lung adenocarcinoma cells with K-RAS mutations (67)

Mehdi et al., 2013)

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