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Ferroptosis describes a form of regulated cell death that is driven by unrestricted lipid peroxidation, which does not require the activation of caspase and can be suppressed by iron chelators, lipophilic antioxidants, inhibitors of lipid peroxidation, and depletion of polyunsaturated fatty acids

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Research suggests that GHK-CU may play a role in collagen synthesis, tissue remodeling, and modulation of inflammation pathways, contributing to cellular repair and regeneration processes