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Since activated phospho-SMAD-2 (p-SMAD2) and p-SMAD-3 are particular intracellular proteins that are involved in the signal transduction of the TGF- pathway, a decrease in their levels is not surprising (Table 2)

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Different handling requirements apply to unreconstituted powder versus reconstituted solution

Although these antidiabetic agents inhibit multiple molecular pathways involved in platelet activation in both preclinical and clinical settings, they fail to completely abolish the persistent platelet dysfunction observed in diabetic patients, which continues to contribute to increased cardiovascular and cerebrovascular risk

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