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These finding imply that targeting MAMs may hold promise as a therapeutic approach for the treatment of neuroinflammatory diseases

These PUFAs are incorporated into phospholipids, making cellular membranes more prone to ferroptosis due to their high susceptibility to lipid peroxidation (47)

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Under normal unstressed conditions, KEAP1 binds to NRF2 and facilitates its degradation via the ubiquitinproteasome pathway, which is crucial for maintaining NRF2 at low levels [77]

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