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Description
This deficit in mitochondrial energy reserve is well-documented in clinical literature [1]
It enhances amino acid uptake and protein synthesis in muscle tissues, increasing muscle growth and repair
Cellular uptake assay To evaluate cellular uptake efficiency and distribution, Cy5-labeled miRNA was employed in 4T1 and B16F10 cells through flow cytometry and confocal laser scanning microscopy (CLSM)
GH treatment (0.4 mg/d for 7 days) of endothelial progenitor cells from patients with atherosclerosis reversed age-related dysfunction and attenuated senescence (indicated by increased telomerase activity) [107]
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