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This functional disorder not only disrupts the normal regulatory mechanisms of blood vessels but also leads to increased blood pressure, arterial stiffness, and remodeling of small arteries, ultimately resulting in multi-organ damage (Boutouyrie et al., 2021

Cysteine is found in the lowest concentrations of the trio required to make GSH

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Less problematic in this respect are the antagonists of the pro-excitotoxic adenosine A 2A receptor [271] as well as drugs directed towards the relatively disregarded low expressed adenosine A 3 receptor [272, 273]

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