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Glutamine activates the extracellular signal-regulated kinases (ERKs) and the c-Jun N-terminal kinases (JNK) in the enterocyte, and it leads to significant increase in c-Jun gene expression and in the activity of the transcription factor known as activator protein 1 (AP-1) [41,42]

Aortic plasma samples revealed lower MOTS-c levels in patients with ED compared to those with normal endothelial function

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