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, the covalent binding of NAPQI to the thiol groups of critical cellular proteins (following glutathione depletion), oxidative stress induced directly by NAPQI or via coproduction of reactive oxygen species (superoxide radical and hydrogen peroxide) through redox cycling between the paracetamol semiiminoquinone radical and NAPQI) (1, 2), and vice versa, these processes counteraction may be involved in BPC 157 beneficial effects

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Then, once in the extracellular space, GSH is cleaved by -Glutamyl-Transferase (GGT), which is also known as a poor prognostic factor for cancer patients (34)

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As always, follow the suggested use instructions and consume the amounts recommended on the label